Comment news, articles, and features | New Scientist /section/comment/ Science news and science articles from New Scientist Fri, 10 Jul 2026 10:02:37 +0000 en-US hourly 1 https://wordpress.org/?v=7.0.1 242057827 The sneaky maths trick for solving problems without answering them /article/2532687-the-sneaky-maths-trick-for-solving-problems-without-answering-them/?utm_campaign=RSS|NSNS&utm_content=comment&utm_medium=RSS&utm_source=NSNS Fri, 10 Jul 2026 08:00:57 +0000 /?post_type=article&p=2532687 2532687 Does time come from the entire universe running computations? /article/2532871-does-time-come-from-the-entire-universe-running-computations/?utm_campaign=RSS|NSNS&utm_content=comment&utm_medium=RSS&utm_source=NSNS Tue, 07 Jul 2026 17:00:36 +0000 /?post_type=article&p=2532871 2532871 5 things to know about sunscreen, according to a skin cancer expert /article/2532744-5-things-to-know-about-sunscreen-according-to-a-skin-cancer-expert/?utm_campaign=RSS|NSNS&utm_content=comment&utm_medium=RSS&utm_source=NSNS Mon, 06 Jul 2026 13:00:27 +0000 /?post_type=article&p=2532744
Sunscreen protects your skin, but just how much do you need?
Shutterstock

When the sun is out, many of us reach for sunscreen, but myths and misinformation have left some people confused about when and how to use it, and how to ensure we still get enough vitamin D. , a skin cancer expert at QIMR Berghofer Medical Research Institute in Australia, has led clinical trials looking at the link between sun exposure and skin cancer, and sunscreen use and vitamin D. She also recently led the development of a new that considers how to balance the risks and benefits of sun exposure. Here, she lists five things that everyone should know about sunscreen.

Sunscreen should be used as a last line of defence

Many people think it’s OK to lie on the beach all day in a tiny bikini as long as they rub sunscreen all over their exposed skin and reapply it every 2 hours. They think sunscreen makes them bulletproof. But even if you apply the best sunscreen perfectly, it still lets some ultraviolet (UV) radiation through. If you’re out in the sun for hours, that gradually adds up to a dose that is big enough to cause skin damage. At that point, it doesn’t matter if you apply more sunscreen; the damage is already done.

People often assume that if they reapply sunscreen every 2 hours, they’re sort of starting the protection again, but that’s not how it works. You also need to protect yourself with a hat, sunglasses, rash shirt or other protective clothing, and stay in the shade in the middle of the day. Sunscreen should be considered a last line of defence for the parts of your skin that you can’t easily cover while you’re outdoors, like your hands and neck.

There is good evidence that sunscreen protects against skin cancer and wrinkles

The largest, longest-running study of sunscreen was conducted in the Australian town of Nambour. In 1992, 1600 people in the town were randomised to apply daily sunscreen or continue with their normal sunscreen use, which tended to be minimal. It found that those who applied the daily sunscreen were years down the track.

The researchers also created moulds of the backs of the study participants’ hands to look at damage to the surface of the skin. Those in the daily sunscreen group had compared with those who didn’t. When they were followed up on in 2014, they also had .

The sunscreen to choose is the one you like wearing

It’s no good having sunscreen that sits in your cupboard and doesn’t end up on your skin because you don’t like the feel of it. If you’re going on a hike and you’re going to be out all day, it’s better to wear sunscreen with a high sun protection factor (SPF) of 50+. But it’s harder to get a high-SPF sunscreen that feels really nice, so if you’ll be popping out for only short periods throughout the day, you can choose an SPF 15 or 30 sunscreen. Tinted sunscreens can offer the same protection as normal sunscreens, but only if you apply them thickly. But because these often make the skin look overly tinted, people tend to apply them too thinly. One option is to first put on a thick layer of normal sunscreen, then apply the tinted sunscreen on top of it.

Chemical sunscreens, meaning those that contain organic ingredients such as octocrylene and avobenzone, work by absorbing UV radiation from the sun and converting it to harmless heat. Inorganic sunscreens, also known as mineral or physical sunscreens, contain zinc oxide or titanium dioxide particles. They are often reported to work by reflecting or scattering UV radiation, but they actually , like chemical sunscreens.

Wearing two layers of sunscreen helps to achieve adequate coverage

You get the SPF listed on the bottle only if you apply 2 milligrams of sunscreen per square centimetre of skin, which is around in an average adult. But it’s really hard to apply this amount of sunscreen in one go. One day, I decided to measure it out exactly, and I couldn’t rub it all on; it was too much. So now, I apply one layer, let it sink in while I brush my teeth and do other things, and then a second layer, so I can apply the full recommended amount.

I was born in Armidale, Australia, in the late 1960s and didn’t wear sunscreen as a child, despite my pale skin. I’ve since had three skin cancers removed, the first of which appeared when I was just 29. So now, I am careful to protect my skin.

If you’re diligent with sunscreen, you might need to take a vitamin D supplement

We recently conducted a trial called the Sun-D Study to see whether applying SPF 50+ sunscreen every day affects people’s vitamin D levels. We randomly assigned 639 people to apply SPF 50+ sunscreen as part of their daily morning routine on days when the UV index was forecast to reach 3 or higher, or to use it at their own discretion. After about a year, a – about 46 per cent compared with 37 per cent in the control group. If you wear sunscreen every day, I would advise taking a vitamin D supplement so that you don’t become deficient, especially in winter. I take one myself – they are cheap, safe and effective.

People with dark-coloured skin are at greater risk of developing a vitamin D deficiency. I recently led the development of a new that looked at how to balance the various risks and benefits of sun exposure. It brought together experts from many Australian universities and medical organisations, and it concluded that people with dark-coloured skin need to put on sunscreen only if they plan to spend more than 2 hours outdoors on days with high UV radiation levels. This is in recognition of the fact that melanoma incidence is 30 times lower in people with dark-coloured skin than in those with light-coloured skin, and that vitamin D deficiency poses a greater risk.

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Beetroot juice is trending – its benefits go beyond the hype /article/2532642-beetroot-juice-is-trending-its-benefits-go-beyond-the-hype/?utm_campaign=RSS|NSNS&utm_content=comment&utm_medium=RSS&utm_source=NSNS Mon, 06 Jul 2026 08:00:36 +0000 /?post_type=article&p=2532642 2532642 Synthetic biology may finally be ready to solve life’s biggest mystery /article/2532794-synthetic-biology-may-finally-be-ready-to-solve-lifes-biggest-mystery/?utm_campaign=RSS|NSNS&utm_content=comment&utm_medium=RSS&utm_source=NSNS Thu, 02 Jul 2026 14:38:05 +0000 /?post_type=article&p=2532794
The synthetic SpudCell shows many of the properties of life
Orion Venero, Adamala Lab
A living organism is made from components that aren’t themselves living. This simple statement has profound implications. For one, it means that there is no mystical force that animates us and other life forms. For another, it means that it should be possible to build a life form from scratch – and we are now a step closer to doing so. Artificial life has been the guiding light of synthetic biology for some time. In 2010, biologists at the J. Craig Venter Institute in California synthesised the stripped-down genome of a bacterium and inserted it into the chassis of another cell, emptied of its own DNA. The resulting organism, with a record-low number of genes (473) was able to grow and reproduce. But even then, scientists didn’t understand what a third of those genes were doing, or whether they were even needed. Instead of rebooting an existing cell with a synthetic genome, we need to build an organism from the ground up. That is what scientists at the University of Missouri are now attempting. The SpudCell – named both to evoke Sputnik and the dawn of the space age, and for its resemblance to a potato – is an entity based on just 36 genes. It self-assembled when the genes were supplied with all the building blocks necessary for life, forming cell-like bubbles and making proteins.

SpudCell represents a significant breakthrough in the creation of artificial life

But that’s it. The SpudCell can only make proteins because it is supplied with ribosomes, the crucial cell components that make proteins. It can’t metabolise food, supply itself with energy or reliably divide and reproduce. It isn’t alive, and it needs intensive care just to perform its basic functions. Nevertheless, the SpudCell represents a significant breakthrough in the creation of artificial life. If a modern living cell is a jet airliner, the SpudCell is the rickety wooden-and-cotton proto-airplane made by the Wright brothers. Better versions will soon follow, with potentially transformative applications. The hope is that synthetic cells will one day be able to supply materials that are currently derived from fossil fuels, such as plastics, fuels and fertiliser. That is keenly needed. But the work in understanding how a living entity operates will shed light on what life needs, and how it emerges from dead materials. If we crack this ultimate mystery, synthetic biology will have really delivered.]]>
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Can climate change stay in the news agenda after Europe’s heatwave? /article/2532443-can-climate-change-stay-in-the-news-agenda-after-europes-heatwave/?utm_campaign=RSS|NSNS&utm_content=comment&utm_medium=RSS&utm_source=NSNS Wed, 01 Jul 2026 18:00:00 +0000 http://mg27136022.400 A person shields their face from the heat of the sun with a fan, in central London on June 25, 2026, during a heatwave. The UK recorded its hottest ever June temperature on June 24 with the mercury rising to 36.1C in southern England, breaking the previous record of 35.6C set in 1976. (Photo by Brook Mitchell / AFP via Getty Images)

The heatwave that swept Europe last week saw many temperature records broken, leading people to ask if extraordinary June heat is the “new normal”. Unfortunately, the truth is that we are never going to have normal in our lifetimes again – just ever more extreme heat.

Climate scientists are continually warning of the need to prepare for hotter heatwaves, worse droughts, more flooding and rising seas. During heatwaves like the one just passed, the hottest and most humid ever seen in Europe, they might even get a little media coverage. But then the weather cools, the news agenda moves on and nothing is ever done.

We are currently on course for average global surface temperatures to rise by between 2.1°C and 3.3°C by 2100, and possibly even more. Even these alarming numbers are a little misleading because the oceans that cover most of the planet don’t warm as fast as the land. Average land temperatures are therefore going to go up by a lot more than the above numbers imply.

But what really matters to us is extreme weather, not the average. The projections for future extremes are already dire, and there are reasons to think that we are in for extremes even greater than those currently projected for a given level of warming. With uncertainties over the survival of the vital AMOC ocean current and the risk of a major glacier collapse, the only thing we really do know is that we must prepare for conditions far worse than Europe has just experienced.

We are on course for average global surface temperatures to rise by between 2.1°C and 3.3°C

It is possible to get through even worse heatwaves if all your infrastructure and systems are geared up to cope, but for most countries, this isn’t the case. The fact is, the world is changing fast and we need to change just about every aspect of our lives to adapt – our homes and offices, factories and schools, cars and trains, farms and gardens, and so on. But it isn’t happening. One day, after the tragic deaths grow too great to bear, we will ask why we did nothing to prevent them.

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I have a 100 per cent chance of getting cancer due to a rare gene /article/2532073-i-have-a-100-per-cent-chance-of-getting-cancer-due-to-a-rare-gene/?utm_campaign=RSS|NSNS&utm_content=comment&utm_medium=RSS&utm_source=NSNS Fri, 26 Jun 2026 14:00:34 +0000 /?post_type=article&p=2532073
Tracy Hutchinson has a rare mutation of the TP53 gene
Tracy Hutchinson
I started to wonder if something funky was going on when multiple people in my family got cancer around the same time. In 1990, my older sister Rebecca was diagnosed with acute lymphoblastic leukaemia, when she was 21 and I was 14. While she was undergoing rigorous chemo, my mum was diagnosed with breast cancer. Rebecca passed away in 1994 and then, a couple of years after that, my dad got bowel cancer. While he was undergoing treatment, my mum got cancer in her other breast. She survived that, but then she was diagnosed with oesophageal cancer in 2009. She had major surgery, but it came back and she died six weeks later. In 2020, my other sister was diagnosed with fast-growing triple-negative breast cancer and I thought, oh my god, there’s something going on here. My sister was tested for the BRCA mutations, variants of the BRCA1 and BRCA2 genes that increase breast cancer risk, and it came back negative. So then she was tested for a different mutation in a gene called TP53, which is much rarer but even worse. Women with this mutation have , with a 50 per cent chance before the age of 30. It’s called Li-Fraumeni syndrome and it basically means your TP53 gene, which normally functions as a cancer-suppressing gene, is a dud. When my sister was offered the test, I was like: “What’s Li-Fraumeni syndrome?” It’s not something you ever hear about. The test came back positive and she was extremely distraught. Since it can run in families, I was then offered the test too. I decided to do it because I didn’t want my sister to go through this journey on her own. I had the test in 2022, when I was 47, and it came back positive. I actually felt at peace with it, which a lot of people find surprising, but it was because I finally felt like I had answers for all the troubles my family has been through. It’s a personal thing, though – my brother, for example, has chosen not to get tested.
After getting the diagnosis, my life changed forever. When you have Li-Fraumeni syndrome, there isn’t an hour where you don’t think about it. It’s always on your mind. Within months of finding out I had the gene, I had a double mastectomy as a preventative measure. They found two ductal carcinomas in situ, early forms of cancer, in my left breast after it was removed. I live in Sydney, so I was able to join an that is investigating annual whole-body MRI as a way to spot tumours in any location in people with mutations in TP53 or other genes that can cause cancer in multiple organs. I had my first one in 2022 and I was very nervous because I didn’t know if they’d find anything. It was normal, but in the second year, they found a 9-millimetre meningioma – a tumour in the meninges, the layers of tissue covering the brain. Fortunately, it’s benign, but I was very freaked out about it. It was a bit of a gamechanger for me. I have my annual whole-body MRI every November and my “scanxiety” starts building from around July. I start thinking, is this going to be the year when everything changes? But being part of the study also gives me a sense of reassurance, because it’s designed to pick up cancers at an early stage when they are hopefully still treatable. My sister, who survived her breast cancer, now has annual MRIs too. In addition to whole-body MRI, I have yearly skin checks with a dermatologist and an annual blood test. Every two years, I also have an endoscopy and colonoscopy. They’ve found polyps, abnormal cell growths that can develop into cancer, in my bowel, which were removed, plus some atypical cells in my oesophagus, which they’re keeping an eye on. I’m also on constant alert for anything unusual in my body. I’ll have a sore shoulder and get nervous because I’ll wonder, is this going to be something? My geneticist thinks my mum might have had a de novo mutation, a mutation that arises spontaneously in an individual rather than being inherited, in her TP53 gene, which was passed down to me and my sisters. We don’t have children, so there isn’t a risk of us passing it on further. My partner has been really supportive. After I found out I had the syndrome, he said: “You’ve just got to do what you’ve got to do.” When I had my double mastectomy, I didn’t go down the path of having a breast reconstruction and I was worried I looked like a freak, but he said: “Not at all. Your scars tell your battle.” I try to stay positive because I figure that everyone has something they’re dealing with, be it a chronic disease or an injury or depression, and this is just my thing to bear. My sister-in-law, for example, recently had a stroke. We all have our things – some are visible and some are not – so we need to be compassionate towards each other. Life isn’t a white picket fence. As told to Alice Klein]]>
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Can video games help us better understand quantum mechanics? /article/2532015-can-video-games-help-us-better-understand-quantum-mechanics/?utm_campaign=RSS|NSNS&utm_content=comment&utm_medium=RSS&utm_source=NSNS Fri, 26 Jun 2026 08:00:02 +0000 /?post_type=article&p=2532015 2532015 Screwworm could be the first species targeted by an ‘extinction drive’ /article/2531859-screwworm-could-be-the-first-species-targeted-by-an-extinction-drive/?utm_campaign=RSS|NSNS&utm_content=comment&utm_medium=RSS&utm_source=NSNS Wed, 24 Jun 2026 17:19:04 +0000 /?post_type=article&p=2531859 2531859 Neuroscience can’t tell us the way to govern people’s brains /article/2531470-neuroscience-cant-tell-us-the-way-to-govern-peoples-brains/?utm_campaign=RSS|NSNS&utm_content=comment&utm_medium=RSS&utm_source=NSNS Wed, 24 Jun 2026 18:00:00 +0000 http://mg27036012.800 Human brain. Digitally enhanced 3D magnetic resonance imaging (MRI) scan of a normal human brain. An eyeball is shown at lower left (blue).

Our brains love shortcuts. Decisions are often made via a subconscious muddling through, due to the . It is perhaps why we value neat categorisations of someone’s brain state, despite these being flawed.

Take the age at which you become an adult. Around the world, legal adulthood varies from 16 to 21. This difference matters, as we rightly have different expectations for children versus adults. Some call for this tension to be smoothed by asking policy-makers to consider typical brain maturity levels, ascertained via tools like brain imaging, in matters like criminal sentencing or the right to drive. The idea that our brains don’t fully develop until we are 25 is also becoming popular, but – as we discuss in our special on brain changes – this is wrong. Brains mature at different rates and there are myriad ways to measure their development.

This isn’t the only way neuroscience is looked to for informing policy before the science is ready. Take autism, which may come in several distinct types. A recently proposed category of “profound autism” could identify those with the highest needs, by assessing IQ, language skills and care requirements. This could assist in advocating for services for people in this group, but may exclude those who don’t meet strict criteria. It could also people with speech difficulties with people with cognitive impairments – different neurological profiles demanding different help.

Neuroscience is looked to for informing policy before the science is ready

Attempts to use psychological profiling in courts, too, are worrying. When presented as a mitigating factor in some cases, , taking a slam-dunk prosecution down a messier road. But . shows that, while potentially legally relevant, it can’t be used with confidence.

Our wish to put brains into tidy boxes is natural, and a future in which neuroscience can help us put a fine point on someone’s cognitive state could well be possible. That future, though, is not yet here.

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